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Cystic fibrosis research paper thesis

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Cystic Fibrosis is one of the most widespread genetic disorders in the modern world. While only 30, members of the American population is affected by the disease, millions of Americans are carriers of the disease; the difference between carriers and sufferers of the disease lies in the autosomal recessive nature of Cystic fibrosis. It was not until recently that scientists learned that the disease arises from a mutation in the CF gene located on chromosome 7. In a person with Cystic fibrosis, the lack of a properly functioning CFTR protein causes for the production of sticky mucus that is excessively thick and very salty sweat. This mucus can then either clog the airways, causing progressive damage and complications with the respiratory system, or can also block ducts in the pancreas, which basically causes vitamin deficiency and malnutrition.
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Cystic fibrosis research paper thesis
Cystic fibrosis research paper thesis
Cystic fibrosis research paper thesis
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Cystic fibrosis is a chronic, inherited, life threatening disease that affects organs in the body, because of sticky and thick mucus buildup on organs. The organs that are affected are the liver, lungs, pancreas, and intestine, which does damage to the respiratory, digestive and reproductive systems Crosta. Cystic fibrosis is caused by a mutation in a gene called cystic fibrosis trans-membrane regulator, also called CFTR, which has an important function of creating sweat, mucus and digestive juices. Cystic Fibrosis CF is an inherited and obstructive lung disease caused by the overall obstruction of the airways with mucus that form in the lungs, pancreas and sweat glands. CF therefore affects the ability to breath and ultimately kills it victims at a young age. Most individuals who have Cystic Fibrosis become progressively worse, and many die in their 20s and 30s.
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Can viruses used as vectors in the process of gene therapy be an effective treatment for Cystic Fibrosis? Firstly, Cystic Fibrosis is a genetic and recessive disease that mostly affects the lungs and pancreas. This leads to phlegm accumulation, salty sweat, male infertility, shortness of breath and increased risk of infection, which all contribute to premature death CFF, Cystic Fibrosis is a disease that forces a person to drown in mucus that fills their lungs while it wreaks havoc on the body.
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Without the cystic fibrosis variation of the CFTR gene, the CFTR proteins created by the gene act as a channel protein which can be found in the membranes of cells which line the passageways of organs such as the pancreas, lungs, and intestines. The CFTR protein can be modified in numerous ways to give the host cystic fibrosis; in fact, over transformations of the CFTR gene have been recognized. One of the most common of these mutations is a deletion of a single amino acid from the long chain of in the CFTR protein. This causes a breakdown of the channel made with the missing amino acid, which means that it never transports chloride ions, like it was made to do, because it never reaches the cell membrane. This order chooses the way that the protein is folded, and if this order is changed or altered, the way that the protein will also be modified, causing the protein to not be able to work.
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